Author

A. Mikhailova

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Assay:proteomics· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:comparative / phylogenetic analysis· 1

Frequent Co-Authors

A. David· 1 shared A. Elfidha· 1 shared A. Saez-Cirion· 1 shared A. Sakuntabhai· 1 shared B. Raynal· 1 shared C. Pereira-Bittencourt Passaes· 1 shared C. Tamietti· 1 shared E. Dohmen· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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1 shared paperMicrobiology

A. David

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Major changes in domain arrangements are associated with the evolution of termite castes

A. Mikhailova, E. Dohmen, M. C. Harrison

The paper investigates domain rearrangements in termite proteomes to understand their role in the evolution of eusociality, finding novel arrangements linked to longevity and caste-specific functions.

bioRxiv · May 15, 2023 · PreprintEvolutionary Biology

The dengue virus NS1 protein conveys pro-inflammatory signals by docking onto human high-density lipoproteins

S. Benfrid, K.-H. P. Park, M. Dellarole, J. E. Voss, C. Tamietti, G. Pehau-Arnaudet, B. Raynal, S. Brule, P. England, X. Zhang, A. Mikhailova, M.-N. Ungeheuer, S. Petres, S. Biering, E. Harris, , M. Flamand

This study identifies NS1-dengue virus protein's interaction with human HDL as a mechanism to trigger pro-inflammatory signals during infection.

bioRxiv · May 5, 2021Microbiology

Anti-apoptotic clone 11 derived peptides induce in vitro death of CD4+ T cells susceptible to HIV-1 infection

A. Mikhailova, J. C. Valle-Casuso, A. David, V. Monceaux, S. Volant, C. Pereira-Bittencourt Passaes, A. Elfidha, M. Muller-Trutwin, J. L. Poyet, A. Saez-Cirion

The paper shows that AAC-11-derived peptides induce death of HIV-1 susceptible CD4+ T cells by targeting metabolically active cells.

bioRxiv · Apr 6, 2020Microbiology