Author

Angelika Amon

Department of Biology, David H. Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02139

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Assay:proteomics· 1 Approach:wet-lab / biochemical assay· 1

Frequent Co-Authors

Colleen M. Meehl· 1 shared Erica S. Chung· 1 shared Jason Meyer Sheltzer· 1 shared Julie H. Ko· 1 shared Nicole C. Habibe Burgos· 1 shared Shannon Y. WengiD· 1 shared Stephen P. BelliD· 1 shared Verena Passerini· 1 shared

Activity by Year

2016-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

Colleen M. Meehl

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2 papers

Recent·Most cited

A noncanonical GTPase signaling mechanism controls exit from mitosis in budding yeast

Xiaoxue Zhou, Shannon Y. Weng, Stephen P. Bell, Angelika Amon

This study reveals a noncanonical GTPase signaling mechanism in budding yeast where Tem1 localization, not its nucleotide state, controls MEN activation by increasing effective concentration for Cdc15 interaction.

bioRxiv · May 16, 2024Cell Biology

Single-chromosome aneuploidy commonly functions as a tumor suppressor

Jason Meyer Sheltzer, Julie H. Ko, Nicole C. Habibe Burgos, Erica S. Chung, Colleen M. Meehl, Verena Passerini, Zuzana Storchova, Angelika Amon

This study shows that single-chromosome aneuploidy often suppresses tumorigenesis, as aneuploid cell lines grew poorly compared to euploid ones, but some evolved to overcome this by acquiring additional karyotype changes.

bioRxiv · Feb 19, 2016Cancer Biology