Author

Annie Tam

BC Cancer Agency

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Peter C. StirlingiD· 2 shared Anissa DjedidiD· 1 shared Arun KumariD· 1 shared Eric BarekeiD· 1 shared Fjodor MerkuriiD· 1 shared Jacek MajewskiiD· 1 shared Jennifer L. FishiD· 1 shared Kym M. BoycottiD· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersMolecular Biology

Peter C. StirlingORCID

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2 papers

Recent·Most cited

Cdc48 regulates intranuclear quality control sequestration of the Hsh155 splicing factor

Veena Mathew, Arun Kumar, Yangyang Kate Jiang, Kyra West, Annie Tam, Peter C. Stirling

This study shows that Cdc48 regulates the deposition of the splicing factor Hsh155 at intranuclear quality control sites and affects its stability and splicing function.

Journal of Cell Science · Nov 10, 2020Peer-reviewedMolecular Biology

Mis-splicing of Mdm2 leads to increased P53-activity and craniofacial defects in a MFDM Eftud2 mutant mouse model

M.-C. Beauchamp, Anissa Djedid, Eric Bareke, Fjodor Merkuri, Rachel Aber, Annie Tam, Matthew A. Lines, Kym M. Boycott, Peter C. Stirling, Jennifer L. Fish, Jacek Majewski, Loydie Anne Jerome‐Majewska

This study shows that EFTUD2 mutations cause mis-splicing of Mdm2, leading to increased P53 activity and craniofacial defects, which can be mitigated by P53 inhibitors.

bioRxiv · Sep 22, 2020 · PreprintDevelopmental Biology