Author

Anoosha Sri

Indiana University Bloomington

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Hui‐Chen LuiD· 2 shared Sen YangiD· 2 shared Zhen‐Xian NiouiD· 2 shared Andrea EnriqueziD· 1 shared Caliel D. Hines· 1 shared Chia‐Shan WuiD· 1 shared Hugo Rodriquez· 1 shared Jason M. TennesseniD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersNeuroscience

Hui‐Chen LuORCID

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2 papers

Recent·Most cited

NAD+ reduction in glutamatergic neurons triggers fatty acid catabolism and neuroinflammation in the brain, mitigated by SARM1 deletion

Zhen‐Xian Niou, Sen Yang, Andrea Enriquez, Anoosha Sri, Caliel D. Hines, Jason M. Tennessen, Chia‐Shan Wu, Jui-Yen Huang, Hui‐Chen Lu

The paper shows that loss of NMNAT2 in glutamatergic neurons causes metabolic shifts and neurodegeneration, which are mitigated by SARM1 deletion.

bioRxiv · May 5, 2025 · PreprintNeuroscience

NMNAT2 in cortical glutamatergic neurons exerts both cell and non-cell autonomous influences to shape cortical development and to maintain neuronal health

Zhen‐Xian Niou, Sen Yang, Anoosha Sri, Hugo Rodriquez, Jonathan Gilley, Michael P. Coleman, Hui‐Chen Lu

This study shows that NMNAT2 deletion in cortical glutamatergic neurons causes neurodegeneration and disrupts cortical development, with Sarm1 loss partially rescuing these effects.

bioRxiv · Feb 5, 2022 · PreprintNeuroscience