Author

Anthony C. Verbeck

Washington University in St. Louis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Neuroscience· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:mouse· 3 Research Focus:cell type annotation· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Celeste M M. KarchiD· 3 shared Emma DanhashiD· 3 shared Guangming HuangiD· 2 shared Shih‐Feng YouiD· 2 shared Abhirami Iyer· 1 shared Abhirami Kannan IyeriD· 1 shared Andrea S. Díaz‐Pacheco· 1 shared Carlos CruchagaiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersNeuroscience

Celeste M M. KarchORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Ms4a4a loss preferentially remodels microglia with limited, compartment-specific effects across central and peripheral immune compartments in 5xFAD mice

Emma Danhash, Anthony C. Verbeck, Abhirami Iyer, Celeste M M. Karch

bioRxiv · Oct 27, 2026 · PreprintNeuroscience

Ms4a4a loss reprograms amyloid-associated microglia and limits dense-core plaque-associated tau spreading

Emma Danhash, Shao‐Yu Fang, Jacob A. Marsh, Ricardo D’Oliveira Albanus, Anthony C. Verbeck, Guangming Huang, Shih‐Feng You, Erin E. Franklin, Richard J. Perrin, Wade K. Self, David M. Holtzman, Celeste M M. Karch

This study shows that loss of Ms4a4a remodels microglial transcriptional programs and reduces dense-core plaque-associated tau spreading in a mouse model of Alzheimer's disease.

bioRxiv · Jul 27, 2026 · PreprintNeuroscience

Ms4a4a deficiency ameliorates plaque pathology in a mouse model of amyloid accumulation

Emma Danhash, Anthony C. Verbeck, Daniel Western, Andrea S. Díaz‐Pacheco, Grant Galasso, Shih‐Feng You, Guangming Huang, Emma Starr, Nadia Miller, Collin J. Nadarajah, Savannah J Tiemann, Erik S. Musiek, Jasmin Herz, Abhirami Kannan Iyer, John R. Cirrito, , Celeste M M. Karch

This study shows that Ms4a4a deficiency reduces amyloid beta pathology in mice by enhancing Aβ clearance, supported by increased MMP-9 production in microglia and human CSF data.

Alzheimer s & Dementia · Aug 1, 2025Peer-reviewedNeuroscience