Author

B. A. Unger

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cell Biology· 1

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Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. P. Wiita· 1 shared G. Aviles· 1 shared K. Xu· 1 shared M. A. Correia· 1 shared M. Kampmann· 1 shared R. Tian· 1 shared X. Guo· 1 shared Y. Liu· 1 shared

Activity by Year

2019-2019 · peak 1/yr

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9 top collaborators · 36 mutual co-author links

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1 shared paperCell Biology

A. P. Wiita

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1 paper

Recent·Most cited

Mitochondrial dysfunction is signaled to the integrated stress response by OMA1, DELE1 and HRI

X. Guo, G. Aviles, Y. Liu, R. Tian, B. A. Unger, Y.-H. T. Lin, A. P. Wiita, K. Xu, M. A. Correia, M. Kampmann

The paper identifies OMA1, DELE1, and HRI as a pathway that relays mitochondrial dysfunction to the integrated stress response, showing its role in activating eIF2 kinase activity and potential therapeutic relevance.

bioRxiv · Jul 26, 2019Cell Biology