Author

B. H. Habermann

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3 Cancer Biology· 2 Biophysics· 1 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 5 Approach:experimental assay· 5 Approach:statistical model· 3 Organism:bacteria (other)· 2 Research Focus:cell type annotation· 2 Research Focus:metabolism & bioenergetics· 2 Organism:mouse· 2 Approach:biophysical modeling· 1

Frequent Co-Authors

F. Marchiano· 2 shared R. Jain· 2 shared T. Mignot· 2 shared A. Carrier· 1 shared A. Le Bivic· 1 shared A. Vernale· 1 shared B. Giannesini· 1 shared B. Lee· 1 shared

Activity by Year

2019-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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2 shared papersCancer Biology

F. Marchiano

Mutual co-authors in cluster

Author profile →

7 papers

Recent·Most cited

Fatty Acid Metabolism and The Oxidative Stress Response Support Bacterial Predation

R. Jain, N.-H. Le, L. Bertaux, J. Baudry, J. Bibette, Y. Denis, B. H. Habermann, T. Mignot

This study identifies genetic and metabolic adaptations in Myxococcus xanthus that enhance bacterial predation by activating fatty acid metabolism and antioxidant responses.

bioRxiv · Dec 11, 2023Microbiology

MechanoProDB: A Web Based Database for Exploring the Mechanical Properties of Proteins

I. Mesbah, B. H. Habermann, F. Rico

MechanoProDB is a web-based database that curates and provides access to experimental and computational data on protein mechanical properties, enabling exploration of unfolding forces and conformational stability.

bioRxiv · Nov 30, 2023Biophysics

Integrative study of skeletal muscle mitochondrial dysfunction in a murine pancreatic cancer-induced cachexia model

T. Gicquel, F. Marchiano, G. Reyes-Castellanos, S. Audebert, L. Camoin, B. H. Habermann, B. Giannesini, A. Carrier

This study investigates mitochondrial dysfunction in skeletal muscle of a murine pancreatic cancer-induced cachexia model, identifying metabolic defects beyond traditional pathways.

bioRxiv · Dec 28, 2022Cancer Biology

Evolution of mechanisms controlling epithelial morphogenesis across animals: new insights from dissociation - reaggregation experiments in the sponge Oscarella lobularis.

A. Vernale, M. M. Prunster, F. Marchiano, H. Debost, N. Brouilly, C. Rocher, D. Massey Harroche, E. RENARD, A. Le Bivic, B. H. Habermann, C. Borchiellini

The paper investigates epithelial morphogenesis in the sponge Oscarella lobularis using dissociation-reaggregation experiments, revealing conserved molecular mechanisms between sponges and bilaterians.

bioRxiv · Mar 22, 2021Evolutionary Biology

BCL-XL blockage in TNBC models confers vulnerability to inhibition of specific cell cycle regulators

O. Castellanet, F. Ahmad, Y. Vinik, G. B. Mills, B. H. Habermann, J.-P. H. Borg, S. Lev, F. Lamballe, F. Maina

This study shows that blocking BCL-XL makes TNBC cells vulnerable to CDK1/2/4 inhibitors, but not other cell cycle regulators, through synergistic effects on survival and apoptosis pathways.

bioRxiv · Mar 16, 2021Cancer Biology

A Tad-like apparatus is required for contact-dependent prey killing in predatory social bacteria

S. Seef, J. Herrou, P. de Boissier, L. My, G. Brasseur, D. Robert, R. Jain, R. Mercier, E. Cascales, B. H. Habermann, T. Mignot

This study identifies a new type IV filament-like machinery, Kil, involved in contact-dependent prey killing by Myxococcus xanthus, showing its role in motility arrest and prey cell plasmolysis.

bioRxiv · Feb 25, 2021Microbiology

Transcriptome responses associated with carbon and energy flows under high salinity stress suggest the overflow of acetyl-CoA from glycolysis and NADPH co-factor induces high lipid accumulation and halotolerance in Chlorella sp. HS2

J.-H. Yun, M. Pierrelee, D.-H. Cho, U. Kim, J. Heo, D.-Y. Choi, Y. J. Lee, B. Lee, H. Kim, B. H. Habermann, Y. K. Chang, H.-S. Kim

This study identifies transcriptome changes in Chlorella sp. HS2 under high salinity stress, revealing lipid accumulation and halotolerance mechanisms through carbon reallocation and NADPH co-factor regulation.

bioRxiv · Oct 28, 2019Microbiology