Author

T. Gicquel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Carrier· 2 shared G. Reyes-Castellanos· 2 shared A. David· 1 shared A. Gerard· 1 shared A. M. Krais· 1 shared B. Giannesini· 1 shared B. H. Habermann· 1 shared B. Jegou· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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Curved chords = mutual co-authorship

2 shared papersCancer Biology

A. Carrier

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Paracetamol/acetaminophen hepatotoxicity: new markers for monitoring the elimination of the reactive N-Acetyl-p-benzoquinone imine

E. Gorrochategui, M. Le Vee, H. Selmi, A. Gerard, J. Chaker, A. M. Krais, C. Lindh, O. Fardel, C. Chevrier, P. Le Cann, G. W. Miller, R. Barouki, B. Jegou, T. Gicquel, D. Kristensen, A. David

The paper identifies thiomethyl metabolites as new markers to monitor NAPQI elimination after therapeutic use or intoxication.

bioRxiv · Apr 28, 2023 · PreprintPharmacology & Toxicology

Integrative study of skeletal muscle mitochondrial dysfunction in a murine pancreatic cancer-induced cachexia model

T. Gicquel, F. Marchiano, G. Reyes-Castellanos, S. Audebert, L. Camoin, B. H. Habermann, B. Giannesini, A. Carrier

This study investigates mitochondrial dysfunction in skeletal muscle of a murine pancreatic cancer-induced cachexia model, identifying metabolic defects beyond traditional pathways.

bioRxiv · Dec 28, 2022Cancer Biology

Adaptation of redox metabolism in drug-tolerant persister cells is a vulnerability to prevent relapse in pancreatic cancer

N. Abdel Hadi, G. Reyes-Castellanos, T. Gicquel, S. Gallardo-Arriaga, E. Boet, J.-E. Sarry, R. Masoud, J. L. IOVANNA, A. Carrier

This study identifies mitochondrial and redox metabolic reprogramming in drug-tolerant persister cells of pancreatic cancer and shows that targeting redox metabolism can prevent relapse.

bioRxiv · Dec 28, 2022 · PreprintCancer Biology