Author

César Casanova-Canelo

Universidad Mayor

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Andrea Puebla-Huerta· 2 shared Sandra Aurora NiñoiD· 2 shared Alenka LovyiD· 1 shared Ariela Vergara-Jaque· 1 shared Armen ZakarianiD· 1 shared Camila Quezada- Gutierez· 1 shared Camila Quezada-Gutiérrez· 1 shared Catalina González-Seguel· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCell Biology

Andrea Puebla-Huerta

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintDec 8, 2025

Bcl-xL interaction with VDAC1 reduces mitochondrial Ca2+ uptake, allowing the establishment of Therapy-Induced Senescence.

Andrea Puebla-Huerta, Pablo Morgado-Cáceres, Camila Quezada- Gutierez…·J. César Cárdenas

This study identifies Bcl-xL-VDAC1 interaction as a mechanism that reduces mitochondrial Ca2+ uptake, promoting therapy-induced senescence by preventing apoptosis.

DOI 10.64898/2025.12.05.692660

humanexperimental assay

𝕏 PostBluesky

Cell Biologynpj AgingPeer-reviewedFeb 21, 2025

Calcium (Ca2+) fluxes at Mitochondria-ER Contact Sites (MERCS) are a new target of senolysis in Therapy-Induced Senescence (TIS).

Andrea Puebla-Huerta, Hernán Huerta, Camila Quezada-Gutiérrez…·César Cárdenas

This study shows that inhibiting ER-mitochondria Ca2+ flux is a new target for senolysis in therapy-induced senescence.

22 citationstop 2%

humanexperimental assay

𝕏 PostBluesky