Author

Camila Quezada-Gutiérrez

Universidad Complutense de Madrid

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:drug response / drug discovery· 1 Organism:mouse· 1

Frequent Co-Authors

Alenka LovyiD· 1 shared Ana Payo-Payo· 1 shared Andrea Puebla-Huerta· 1 shared Ángela Zarco-CuadrilleroiD· 1 shared Armen ZakarianiD· 1 shared Carmen Hernández· 1 shared Catalina González-Seguel· 1 shared César CárdenasiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

Alenka LovyORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJul 29, 2025

Transient CB2 receptor activation triggers irreversible luminal differentiation via chromatin remodeling in breast cancer

Nuria G. Martínez-Illescas, Gemma Pérez-García, María Rubert-Hernández…·María Salazar‐Roa

The paper shows that transient CB2R activation in breast cancer cells leads to irreversible luminal differentiation through chromatin remodeling, enhancing tamoxifen response and reducing resistance.

DOI 10.1101/2025.07.29.667375

mousehuman

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Cell Biologynpj AgingPeer-reviewedFeb 21, 2025

Calcium (Ca2+) fluxes at Mitochondria-ER Contact Sites (MERCS) are a new target of senolysis in Therapy-Induced Senescence (TIS).

Andrea Puebla-Huerta, Hernán Huerta, Camila Quezada-Gutiérrez…·César Cárdenas

This study shows that inhibiting ER-mitochondria Ca2+ flux is a new target for senolysis in therapy-induced senescence.

22 citationstop 2%

humanexperimental assay

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