Author

C. F. R. Ascencao

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

M. B. Smolka· 2 shared R. Freire· 2 shared A. Dziubek· 1 shared A. Grimson· 1 shared D. Dibitetto· 1 shared E. A. Fogarty· 1 shared J. Badar· 1 shared J. R. Sims· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCell Biology

M. B. Smolka

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2 papers

Recent·Most cited

Cell BiologyeLifePeer-reviewedFeb 23, 2024

A TOPBP1 Allele Causing Male Infertility Uncouples XY Silencing Dynamics From Sex Body Formation

C. F. R. Ascencao, J. Sims, A. Dziubek…·M. B. Smolka

This study identifies a TOPBP1 mutation causing male infertility by disrupting ATR-dependent MSCI dynamics without affecting SB formation.

DOI 10.7554/elife.90887.3

mouseproteomics

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Cancer BiologybioRxivPreprintJan 13, 2020

ATR Inhibitors as Potent Modulators of DNA End Resection Capacity

D. Dibitetto, J. R. Sims, C. F. R. Ascencao…·M. B. Smolka

This study shows that long-term ATR inhibition reduces DNA end resection and DNA repair efficiency in cancer cells, offering a strategy to enhance PARP inhibitor sensitivity.

DOI 10.1101/2020.01.13.905059

humanDNA methylation

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