Author

C. H. Michel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:C. elegans· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Research Focus:protein folding & dynamics· 1 Assay:protein structure· 1

Frequent Co-Authors

A. D. Stephens· 1 shared C. F. Kaminski· 1 shared C. Huang· 1 shared C. Poudel· 1 shared D. C. David· 1 shared G. S. Kaminski Schierle· 1 shared M. C. Lechler· 1 shared M. M. Vendruscolo· 1 shared

Activity by Year

2018-2018 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

A. D. Stephens

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Intrinsically aggregation-prone proteins form amyloid-like aggregates and contribute to tissue aging in C. elegans

C. Huang, S. Wagner-Valladolid, A. D. Stephens, R. Jung, C. Poudel, T. Sinnige, M. C. Lechler, N. Schlörit, R. F. Laine, C. H. Michel, M. M. Vendruscolo, C. F. Kaminski, G. S. Kaminski Schierle, D. C. David

This study shows that age-dependent protein aggregation in C. elegans forms amyloid-like structures and accelerates tissue functional decline, suggesting a role for protein aggregation in aging.

bioRxiv · Sep 14, 2018Cell Biology