Author

C. M. Horvath

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Approach:statistical model· 1

Frequent Co-Authors

G. Alvarado· 1 shared J. J. Lenoir· 1 shared J. L. Mendoza· 1 shared J.-P. Parisien· 1 shared R. Mandhana· 1 shared T. Aung· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 4 mutual co-author links

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1 shared paperMicrobiology

G. Alvarado

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Enhanced Complex Stability and Optimal JAK Geometry are Pivotal for a Potent Type III Interferon Response

T. Aung, C. M. Horvath, J. L. Mendoza

This study identifies that optimizing JAK-JAK geometry enhances type III IFN signaling potency by 2-3 logs, offering insights into cytokine signaling and therapeutic development.

bioRxiv · Sep 27, 2022 · PreprintCell Biology

The Human STAT2 Coiled-Coil Domain Contains a Degron for Zika Virus Interferon Evasion

J.-P. Parisien, J. J. Lenoir, G. Alvarado, C. M. Horvath

This study identifies the STAT2 coiled-coil domain as a target for Zika virus NS5-mediated degradation, revealing a mechanism of interferon evasion and potential antiviral targets.

bioRxiv · Oct 1, 2021Microbiology

Sendai Virus Infection Induces Expression of Novel RNAs in Human Cells

R. Mandhana, C. M. Horvath

This study identifies novel virus-inducible RNAs (nviRNAs) in human cells infected with Sendai virus, influenza virus, or herpes simplex virus 1, suggesting they may function as antiviral factors.

bioRxiv · Oct 24, 2018Immunology