Author

C. M. Ripplinger

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:electrophysiology· 1 Assay:imaging· 1 Organism:mouse· 1

Frequent Co-Authors

A. Guevara· 1 shared B. A. Christiansen· 1 shared B. A. Habecker· 1 shared B. Osipov· 1 shared C. E. R. Smith· 1 shared F. Zhao· 1 shared G. A. Ng· 1 shared I.-J. Lee· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperPhysiology

A. Guevara

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2 papers

Recent·Most cited

Chronic nicotine exposure is associated with electrophysiological and sympathetic remodeling in the intact rabbit heart

A. Guevara, C. E. R. Smith, J. L. Caldwell, L. Ngo, L. R. Mott, I.-J. Lee, S. Tapa, Z. Wang, L. Wang, W. R. Woodward, G. A. Ng, B. A. Habecker, C. M. Ripplinger

This study shows chronic nicotine exposure in rabbits increases heart rate, lowers alternans threshold, and reduces sympathetic responses, likely due to hypo-innervation and diminished β-adrenergic responsiveness.

bioRxiv · Nov 23, 2023Physiology

Systemic Bone Loss Following Myocardial Infarction in Mice is Mitigated by Treatment with a β3 Adrenergic Receptor Antagonist

P. M. Tjandra, M. P. Paralkar, B. Osipov, Y.-J. Chen, F. Zhao, C. M. Ripplinger, B. A. Christiansen

This study shows that myocardial infarction causes systemic bone loss in mice, which is partially prevented by β3-adrenergic receptor antagonist treatment.

bioRxiv · Feb 20, 2020 · PreprintPhysiology