Author

M. P. Paralkar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1

Frequent Co-Authors

A. J. Struck· 1 shared A. Margolin· 1 shared A. Nellore· 1 shared A. Nguyen· 1 shared B. A. Christiansen· 1 shared B. Osipov· 1 shared C. M. Ripplinger· 1 shared F. Zhao· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. J. Struck

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2 papers

Recent·Most cited

Systemic Bone Loss Following Myocardial Infarction in Mice is Mitigated by Treatment with a β3 Adrenergic Receptor Antagonist

P. M. Tjandra, M. P. Paralkar, B. Osipov, Y.-J. Chen, F. Zhao, C. M. Ripplinger, B. A. Christiansen

This study shows that myocardial infarction causes systemic bone loss in mice, which is partially prevented by β3-adrenergic receptor antagonist treatment.

bioRxiv · Feb 20, 2020 · PreprintPhysiology

Population-level distribution and putative immunogenicity of cancer neoepitopes

M. A. Wood, M. Paralkar, M. P. Paralkar, A. Nguyen, A. J. Struck, K. Ellrott, A. Margolin, A. Nellore, R. F. Thompson

The paper introduces four metrics to prioritize neoepitopes based on peptide novelty and evaluates their immunogenicity using TCGA and melanoma patient data.

BMC Cancer · Apr 13, 2018Peer-reviewedCancer Biology