Author

C. Munz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:flow cytometry· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Assay:DNA methylation· 1

Frequent Co-Authors

O. Chijioke· 2 shared A. D. Valencia-Camargo· 1 shared A. Gerbaulet· 1 shared A. Loff· 1 shared A. Murer· 1 shared A. Roers· 1 shared A. Roesen-Wolff· 1 shared B. Chatterjee· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersImmunology

O. Chijioke

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Epstein Barr virus infection induces tissue-resident memory T cells in mucosal lymphoid tissues

D. Kirchmeier, Y. Deng, L. Rieble, F. Laderach, P. Schuhmachers, A. D. Valencia-Camargo, A. Murer, N. Caduff, B. Chatterjee, O. Chijioke, K. Zens, C. Munz

This study shows EBV infection induces tissue-resident memory T cells in mucosal lymphoid tissues in mice with humanized immune systems, but these TRMs fail to control viral loads compared to systemic effector memory T cells.

bioRxiv · Nov 24, 2023Immunology

Human effector CD8+ T cells with an exhausted-like phenotype control tumor growth in vivo in a humanized tumor model

J. Mietz, M. Kaulfuss, L. Egli, L. Opitz, C. Munz, O. Chijioke

This study establishes an immunocompetent humanized tumor model and identifies exhausted-like CD8+ T cells with antitumor activity in vivo.

bioRxiv · Oct 11, 2023 · PreprintImmunology

Clearance of genome-damaged cells from the hematopoietic system via p53 without contribution by the cGAS/STING axis

N. Dressel, L. Natusch, C. Munz, S. Costas-Ramon, M. N. F. Morcos, A. Loff, B. Hiller, H. Luksch, A. Roesen-Wolff, A. Roers, R. Behrendt, A. Gerbaulet

This study shows that p53 clears genome-damaged hematopoietic cells without needing the cGAS/STING pathway, and that cGAS/STING is not required for coping with DNA damage or preventing leukemia in RER-deficient conditions.

bioRxiv · Jun 24, 2022 · PreprintCell Biology