Author

B. Hiller

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Bioengineering· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Organism:mouse· 3 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Assay:DNA methylation· 1

Frequent Co-Authors

A. A. Raslan· 1 shared A. Gerbaulet· 1 shared A. Loff· 1 shared A. Roers· 1 shared A. Roesen-Wolff· 1 shared B. Suki· 1 shared C. Eberman· 1 shared C. Lopez· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperBioengineering

A. A. Raslan

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Crystal ribcage: a platform for probing real-time lung function at cellular resolution in health and disease

R. Banerji, G. N. Grifno, L. Shi, D. Smolen, R. LeBourdais, J. Muhvich, C. Eberman, B. Hiller, J. Lee, K. Regan, S. Zheng, S. S. Zhang, J. Jiang, A. A. Raslan, J. C. Breda, , H. T. Nia

This paper introduces a transparent ribcage for real-time, cellular-resolution imaging of lung function in health and disease, revealing insights into tumor progression, immune responses, and disease remodeling.

bioRxiv · Oct 28, 2022 · PreprintBioengineering

Clearance of genome-damaged cells from the hematopoietic system via p53 without contribution by the cGAS/STING axis

N. Dressel, L. Natusch, C. Munz, S. Costas-Ramon, M. N. F. Morcos, A. Loff, B. Hiller, H. Luksch, A. Roesen-Wolff, A. Roers, R. Behrendt, A. Gerbaulet

This study shows that p53 clears genome-damaged hematopoietic cells without needing the cGAS/STING pathway, and that cGAS/STING is not required for coping with DNA damage or preventing leukemia in RER-deficient conditions.

bioRxiv · Jun 24, 2022 · PreprintCell Biology

Fibroblast growth factor-9 expression in airway epithelial cells amplifies the type I interferon response and alters influenza A virus pathogenesis

B. Hiller, Y. Yin, Y.-C. Perng, I. de Araujo Castro, L. Fox, M. Locke, K. Monte, C. Lopez, D. Ornitz, D. Lenschow

This study shows that FGF9 overexpression in airway epithelial cells increases type I interferon response and leads to more severe influenza A virus infection and mortality.

bioRxiv · Dec 22, 2021Immunology