Author

C. Rodriguez Rodrigues

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Assay:flow cytometry· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. C. Cumino· 1 shared A. Mendes· 1 shared A. W. Paton· 1 shared A.-M. Lennon-Dumenil· 1 shared C. Almeida· 1 shared D. Barros· 1 shared D. Sanseau· 1 shared E. Gatti· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperImmunology

A. C. Cumino

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Author profile →

2 papers

Recent·Most cited

Modulation of the mTOR pathway plays a central role in dendritic cell functions after Echinococcus granulosus antigen recognition.

C. Rodriguez Rodrigues, M. C. Nicolao, M. Chop, N. Pla, M. Massaro, J. Loos, A. C. Cumino

This study investigates how Echinococcus granulosus antigens modulate dendritic cell functions via the mTOR pathway, affecting cytokine production, co-stimulatory molecules, and antigen presentation.

bioRxiv · Jul 27, 2020Immunology

Developmentally regulated PERK activity renders dendritic cells insensitive to subtilase cytotoxin-induced integrated stress response

P. Pierre, A. Mendes, J. P. Gigan, C. Rodriguez Rodrigues, S. A. Choteau, D. Sanseau, D. Barros, C. Almeida, V. Camosseto, L. Chasson, A. W. Paton, J. C. Paton, R. J. Argüello, A.-M. Lennon-Dumenil, E. Gatti

The paper shows dendritic cells have high eIF2 phosphorylation due to PERK activation, but this does not lead to chronic ISR or translation arrest, allowing them to adapt to stress.

bioRxiv · Apr 27, 2020 · PreprintImmunology