Author

C. Sepich-Poore

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 2 Approach:experimental assay· 2 Assay:metagenomics· 2 Research Focus:cell type annotation· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1 Organism:mouse· 1

Frequent Co-Authors

C. He· 2 shared A. C. Zhu· 1 shared A. Sanchez Castillo· 1 shared E. Schmitt· 1 shared J. Wei· 1 shared K. Wen· 1 shared L. Zhang· 1 shared Q. Dai· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

C. He

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2 papers

Recent·Most cited

The mechanism underlying redundant functions of the YTHDF proteins

Z. Zou, C. Sepich-Poore, X. Zhou, J. Wei, C. He

This study investigates the redundant functions of YTHDF proteins, showing that YTHDF1 and YTHDF2 have distinct roles in RNA decay and condensate formation, and that RNA stabilization after YTHDF depletion is m6A-independent.

bioRxiv · May 5, 2022Cell Biology

The METTL5-TRMT112 N6-methyladenosine methyltransferase complex regulates metabolism and development via translation

C. Sepich-Poore, Z. Zheng, E. Schmitt, K. Wen, Z. S. Zhang, X.-L. Cui, Q. Dai, A. C. Zhu, L. Zhang, A. Sanchez Castillo, X. Zhuang, C. He, S. Nachtergaele

This paper identifies the METTL5-TRMT112 complex as the methyltransferase responsible for m6A modification at 18S rRNA, linking it to metabolism and development.

bioRxiv · May 18, 2021Biochemistry