Author

C. Wouters

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

a. Belot· 1 shared A. Dhir· 1 shared A. Lepelley· 1 shared A. Martin· 1 shared B. B. Manshian· 1 shared B. Bader-Meunier· 1 shared B. Callewaert· 1 shared C. Bodemer· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperImmunology

a. Belot

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Multinucleation resets human macrophages for specialized functions at the expense of mononuclear phagocyte identity

K. Ahmadzadeh, M. Pereira, M. Vanoppen, E. Bernaerts, J.-H. Ko, T. Mitera, C. Maksoudian, B. B. Manshian, S. Soenen, C. D. Rose, P. Matthys, C. Wouters, J. Behmoaras

This study shows that multinucleation in human macrophages reduces mononuclear phagocyte identity and enables specialized functions in multinucleated giant cells.

bioRxiv · Aug 22, 2022Cell Biology

Steroid hormone catabolites activate the pyrin inflammasome through a non-canonical mechanism

F. Magnotti, D. Chirita, S. Dalmon, A. Martin, P. Bronnec, J. Sousa, o. Helynck, W. Lee, D. Kastner, J. J. Chae, M. McDermott, a. Belot, M. Popoff, P. Seve, S. Georgin-Lavialle, , T. Henry

This study identifies steroid hormone catabolites as specific activators of the pyrin inflammasome through a non-canonical mechanism, linking them to autoinflammation and explaining steroid fever.

bioRxiv · Oct 29, 2021Immunology

Enhanced cGAS-STING-dependent interferon signaling associated with mutations in ATAD3A

A. Lepelley, E. Della Mina, E. Van Nieuwenhove, L. Waumans, S. Fraitag, G. I. Rice, A. Dhir, M.-L. Fremond, M. P. Rodero, L. Seabra, E. Carter, C. Bodemer, D. Buhas, B. Callewaert, P. de Lonlay, , Y. J. Crow

The paper identifies mutations in ATAD3A causing enhanced type I interferon signaling in patients with neurological and autoimmune features.

bioRxiv · Apr 2, 2021Immunology