Author

D. B. Foster

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

A. Bhalodia· 1 shared A. Pekosz· 1 shared A. Sidor· 1 shared B. Kim· 1 shared B. O'Rourke· 1 shared C. D. Cohen· 1 shared C. Duque· 1 shared D. Ashok· 1 shared

Activity by Year

2024-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperImmunology

A. Bhalodia

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Author profile →

2 papers

Recent·Most cited

Splenic follicular B cells promote adverse cardiac remodeling after myocardial infarction via MHC II-dependent antigen presentation.

O. V. Ebenebe, J. P. Lovell, C. Duque, Y. Song, S. Rousseau, K. S. Keole, S. Sedighi, A. Bhalodia, K. Bermea, C. D. Cohen, M. R. Mugnier, D. B. Foster, S. Krummey, S. Sidoli, L. Adamo

This study shows splenic follicular B cells worsen heart failure after MI by presenting cardiac antigens via MHC II, promoting adverse cardiac remodeling.

bioRxiv · May 8, 2024 · PreprintImmunology

Innate Immune Activation and Mitochondrial ROS Invoke Persistent Cardiac Conduction System Dysfunction after COVID-19

D. Ashok, T. Liu, J. Criscione, M. Prakash, B. Kim, J. Chow, M. Craney, K. Papanicolaou, A. Sidor, D. B. Foster, A. Pekosz, J. Villano, D.-H. Kim, B. O'Rourke

This study shows that SARS-CoV-2 infection causes persistent cardiac conduction system dysfunction in hamsters, driven by innate immune activation and mitochondrial ROS, with potential implications for long COVID.

bioRxiv · Jan 5, 2024Pathology