Author

L. Adamo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 2 Cell Biology· 1 Microbiology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:mouse· 4 Approach:experimental assay· 3 Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2

Frequent Co-Authors

A. Bhalodia· 3 shared C. D. Cohen· 2 shared C. Duque· 2 shared E. G. Lakatta· 2 shared K. C. Bermea· 2 shared M. D. Zita· 2 shared S. Rousseau· 2 shared V. Kumar· 2 shared

Activity by Year

2022-2024 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersImmunology

A. Bhalodia

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Splenic follicular B cells promote adverse cardiac remodeling after myocardial infarction via MHC II-dependent antigen presentation.

O. V. Ebenebe, J. P. Lovell, C. Duque, Y. Song, S. Rousseau, K. S. Keole, S. Sedighi, A. Bhalodia, K. Bermea, C. D. Cohen, M. R. Mugnier, D. B. Foster, S. Krummey, S. Sidoli, L. Adamo

This study shows splenic follicular B cells worsen heart failure after MI by presenting cardiac antigens via MHC II, promoting adverse cardiac remodeling.

bioRxiv · May 8, 2024 · PreprintImmunology

A murine model of Trypanosoma brucei-induced myocarditis and cardiac dysfunction

N. Crilly, M. D. Zita, A. Beaver, P. Sysa-Shah, A. Bhalodia, K. Gabrielson, L. Adamo, M. Mugnier

This paper develops a reproducible murine model of T. brucei-induced myocarditis and cardiac dysfunction, showing immune-mediated cardiac damage and elevated NT-proBNP levels.

bioRxiv · Oct 5, 2023 · PreprintMicrobiology

Myocardial B cells have specific gene expression and predicted interactions in Dilated Cardiomyopathy and Arrhythmogenic Right Ventricular Cardiomyopathy

K. C. Bermea, C. Duque, C. D. Cohen, A. Bhalodia, S. Rousseau, J. Lovell, M. D. Zita, M. C. Mugnier, L. Adamo

The paper identifies B cell gene expression and interactions in healthy and diseased human myocardium, finding disease-specific changes in DCM and ARVC.

bioRxiv · Sep 21, 2023Immunology

Cardiomyocyte-specific adenylyl cyclase type-8 overexpression induces activation of RelA together with myocardial and systemic inflammation

V. Kumar, K. C. Bermea, D. Kumar, A. Singh, A. Verma, M. Kaileh, R. Sen, E. G. Lakatta, L. Adamo

This study shows that overexpressing adenylyl cyclase type-8 in cardiomyocytes activates RelA and causes myocardial and systemic inflammation, leading to accelerated aging and reduced lifespan.

bioRxiv · Jul 15, 2023 · PreprintCell Biology

A Remarkable Adaptive Paradigm Of Heart Performance And Protection Emerges In Response To The Constitutive Challenge Of Marked Cardiac-Specific Overexpression Of Adenylyl Cyclase Type 8

K. V. Tarasov, K. Chakir, D. R. Riordon, A. E. Lyashkov, I. Ahmet, M. G. Perino, A. J. Silvester, J. Zhang, M. Wang, Y. O. Lukyanenko, J.-H. Qu, M. C.-R. Barrera, M. Juhaszova, Y. S. Tarasova, B. Ziman, , E. G. Lakatta

This study shows that cardiac-specific overexpression of AC8 in mice leads to adaptive cardiac changes resembling cancer-like survival mechanisms without heart failure.

bioRxiv · May 20, 2022Physiology