Author

D. Cikes

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Biophysics· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 4 Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:gene regulatory network inference· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

J. M. Penninger· 2 shared A. Hagelkrueys· 1 shared A. Kautzky-Willer· 1 shared A. Peaucelle· 1 shared B. Lair· 1 shared C. Bergoglio· 1 shared C. Knauf· 1 shared C. Moro· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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2 shared papersPhysiology

J. M. Penninger

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Imaging the microscopic viscoelastic anisotropy in living cells

H. Keshmiri, D. Cikes, M. Samalova, L. Schindler, L.-M. Appel, M. Urbanek, I. Yudushkin, D. Slade, W. J. Weninger, A. Peaucelle, J. Penninger, K. Elsayad

This paper introduces BLAM, a technique to map high-frequency viscoelastic anisotropy in living cells, and applies it to study plant cell wall growth and mammalian nuclear mechanics.

bioRxiv · May 28, 2023 · PreprintBiophysics

The negative adipogenesis regulator DLK1 is transcriptionally regulated by TIS7 (IFRD1) and translationally by its orthologue SKMc15 (IFRD2)

I. Vietor, D. Cikes, K. Piironen, T. Vasakou, D. Heimdoerfer, R. Gstir, M. D. Erlacher, I. Tancevski, P. Eller, E. Demetz, M. Hess, V. Kuhn, G. Degenhart, J. Rozman, M. Klingenspor, , L. A. Huber

This paper identifies two regulatory mechanisms, transcriptional and translational, by which TIS7 and SKMc15 control DLK1 levels, impacting adipogenesis and obesity resistance.

bioRxiv · Apr 3, 2023Cell Biology

Neuropeptide Neuromedin B does not alter body weight and glucose homeostasis nor does it act as an insulin-releasing peptide

D. Cikes, P. J. Atanes, G.-C. Huang, S. J. Persaud, J. M. Penninger

This study investigates the role of Neuromedin B in body weight, glucose homeostasis, and insulin release using genetic models and metabolic analyses.

Scientific Reports · Jun 7, 2022Peer-reviewedPhysiology

Glucose intolerance in aging is mediated by the Gpcpd1-GPC metabolic axis

D. Cikes, M. Leutner, S. J. - F. Cronin, M. Novatchkova, L. Pfleger, R. Klepochova, E. Rullman, T. Gustafsson, A. Hagelkrueys, C. Knauf, C. Moro, M. Krebs, A. Kautzky-Willer, M. Krssak, M. Orthofer, , N. Viguerie

This study identifies Gpcpd1-GPC metabolic pathway perturbation in aging muscle as a driver of glucose intolerance.

bioRxiv · Oct 26, 2021 · PreprintPhysiology