Author

D. Pirri

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

B. Tardajos Ayllon· 2 shared E. Chambers· 2 shared J. Serbanovic-Canic· 2 shared M. Fragiadaki· 2 shared A. Mammoto· 1 shared C. L. Miller· 1 shared C. Souilhol· 1 shared E. Osto· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersPhysiology

B. Tardajos Ayllon

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2 papers

Recent·Most cited

EPAS1 Attenuates Atherosclerosis Initiation at Disturbed Flow Sites through Endothelial Fatty Acid Uptake

D. Pirri, S. Tian, B. Tardajos Ayllon, S. Irving, F. Donati, S. Allen, T. Mammoto, G. Vilahur, C. L. Miller, T. Schenkel, E. Chambers, M. J. Dunning, J. Serbanovic-Canic, F. Botre, A. Mammoto, , P. C. Evans

This study shows EPAS1 reduces atherosclerosis at disturbed flow sites by promoting endothelial fatty acid uptake and metabolism, which is suppressed in obesity through triglyceride-PHD3 pathway.

bioRxiv · Dec 5, 2023Physiology

JAG1-NOTCH4 MECHANOSENSING DRIVES ATHEROSCLEROSIS

C. Souilhol, X. Li, L. Canham, H. Roddie, D. Pirri, B. Tardajos Ayllon, E. Chambers, M. Dunning, M. Ariaans, J. Li, Y. Fang, M. FRAGIADAKI, V. Ridger, J. Serbanovic-Canic, S. De Val, , P. Evans

This study shows JAG1-NOTCH4 signaling promotes atherosclerosis by repressing endothelial repair in response to low oscillatory shear stress.

bioRxiv · May 15, 2020Pathology