Author

D. R. Emlet

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

C. E. Magyar· 1 shared C. L. Kinlough· 1 shared E. K. Jackson· 1 shared J. A. Kellum· 1 shared J. R. Hepler· 1 shared K. E. Squires· 1 shared M. M. Al-bataineh· 1 shared P. A. Friedman· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPharmacology & Toxicology

C. E. Magyar

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

KIM-1-mediated anti-inflammatory activity is preserved by MUC1 induction in the proximal tubule during ischemia-reperfusion injury.

M. M. Al-bataineh, C. L. Kinlough, Z. Mi, E. K. Jackson, S. Mutchler, D. R. Emlet, J. A. Kellum, R. P. Hughey

The paper shows that MUC1 preserves KIM-1's anti-inflammatory activity during kidney injury by preventing its cleavage by ADAM17.

bioRxiv · May 20, 2021 · PreprintPathology

Genetic variants disrupt human RGS14 binding to NHERF1 and regulation of NPT2A-mediated phosphate transport

P. A. Friedman, T. Mamonova, C. E. Magyar, K. E. Squires, W. B. Sneddon, D. R. Emlet, J. R. Hepler

This study identifies how genetic variants in RGS14 affect its interaction with NHERF1 and regulation of phosphate transport, showing some variants disrupt binding while others maintain function.

bioRxiv · Feb 5, 2019 · PreprintPharmacology & Toxicology