Author

D. R. King

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Assay:electrophysiology· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

G. A. Blair· 2 shared G. S. Hoeker· 2 shared R. G. Gourdie· 2 shared R. Veeraraghavan· 2 shared S. Poelzing· 2 shared A. Buxton· 1 shared A. J. Lozano· 1 shared A. L. Hanlon· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersBiophysics

G. A. Blair

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Unraveling Chamber-specific Differences in Intercalated Disc Ultrastructure and Molecular Organization and Their Impact on Cardiac Conduction

H. L. Struckman, N. Moise, D. R. King, A. M. Soltisz, A. Buxton, I. Dunlap, Z. Chen, P. B. Radwanski, S. H. Weinberg, R. Veeraraghavan

This study quantifies intercalated disc structure in mouse heart chambers using advanced imaging and modeling to reveal chamber-specific differences in conduction speed.

JACC: Clinical Electrophysiology · Dec 1, 2023Peer-reviewedPhysiology

EXTRACELLULAR PERINEXAL SEPARATION IS A PRINCIPAL DETERMINANT OF CARDIAC CONDUCTION

W. P. Adams, T. B. Raisch, Y. Zhao, R. Davalos, S. Barrett, D. R. King, C. B. Bain, K. Colucci, G. A. Blair, A. J. Lozano, A. L. Hanlon, R. Veeraraghavan, X. Wan, I. Deschenes, J. Smyth, , S. Poelzing

This study investigates the relationship between cardiac conduction velocity and extracellular space structure, finding a biphasic relationship with perinexal separation, supporting ephaptic coupling over gap junction-based conduction.

bioRxiv · May 25, 2023Biophysics

Acute adenoviral infection elicits an arrhythmogenic substrate prior to myocarditis

R. L. Padget, M. J. Zeitz, G. A. Blair, X. Wu, M. D. North, G. S. Hoeker, K. E. Stanley, C. M. Phillips, D. R. King, R. G. Gourdie, M. T. Tanenbaum, G. S. Hoeker, S. A. Swanger, S. Poelzing, J. W. Smyth

This study shows adenoviral infection causes arrhythmogenic changes in the heart before significant immune response, through gap junction and ion channel dysfunction.

bioRxiv · Nov 29, 2022Pathology