Author

D. Ragusa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

C. Pina· 2 shared A. Curti· 1 shared A. F. Domingues· 1 shared A. Sala· 1 shared A. V. Taylor· 1 shared B. J. Huntly· 1 shared C. Federico· 1 shared C. Sisu· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCancer Biology

C. Pina

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Author profile →

2 papers

Recent·Most cited

MAT2A inhibition in AML unveils therapeutic potential of combining DNA demethylating agents with UPR targeting

K. Zeka, A. V. Taylor, R. Samarista, D. Ragusa, C.-W. Suen, O. W. Cyran, A. F. Domingues, E. Meduri, B. J. Huntly, D. Forte, A. Curti, C. Pina

The paper shows that combining DNA demethylating agents with UPR targeting improves AML cell suppression beyond single-agent therapies.

bioRxiv · Jun 5, 2023 · PreprintCancer Biology

Engineered model of t(7;12)(q36;p13) AML recapitulates patient-specific features and gene expression profiles

D. Ragusa, Y. Ciciro, C. Federico, S. Saccone, F. Bruno, R. Saeedi, C. Sisu, C. Pina, A. Sala, S. Tosi

This study creates a t(7;12) AML model in K562 cells that mirrors patient-specific gene expression and features, aiding in understanding disease mechanisms.

bioRxiv · Jun 14, 2022Cancer Biology