Author

Debora Tibbe

University Medical Center Hamburg-Eppendorf

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Research Focus:protein structure prediction· 2 Assay:proteomics· 2 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Hans‐Jürgen KreienkampiD· 3 shared Daniel WoikeiD· 2 shared Emily Wang· 2 shared Fatemeh Hassani NiaiD· 2 shared Igor BarsukoviD· 2 shared Antonio Virgilio FaillaiD· 1 shared Christina Ringmann FagerbergiD· 1 shared Christoph KrispiD· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersNeuroscience

Hans‐Jürgen KreienkampORCID

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3 papers

Recent·Most cited

The Shank/ProSAP N-terminal (SPN) domain of Shank3 regulates targeting to postsynaptic sites and postsynaptic signalling.

Daniel Woike, Debora Tibbe, Fatemeh Hassani Nia, Victoria Martens, Emily Wang, Igor Barsukov, Hans‐Jürgen Kreienkamp

This study investigates how the SPN-Ank domain of Shank3 regulates its localization and function in postsynaptic sites, showing its role in synaptic signaling and implications for autism-related mutations.

bioRxiv · Apr 28, 2023 · PreprintNeuroscience

Regulation of Liprin-α phase separation by CASK is disrupted by a mutation in its CaM kinase domain

Debora Tibbe, Pia Ferle, Christoph Krisp, Sheela Nampoothiri, Ghayda Mirzaa, Melissa Assaf, Sumit Parikh, Kerstin Kutsche, Hans‐Jürgen Kreienkamp

The paper shows that a mutation in CASK's CaMK domain disrupts its regulation of Liprin-α phase separation, leading to neurodevelopmental disorder with microcephaly and pontocerebellar hypoplasia.

Life Science Alliance · Sep 22, 2022Peer-reviewedGenetics

Mutations affecting the N-terminal domains of SHANK3 point to different pathomechanisms in neurodevelopmental disorders.

Daniel Woike, Emily Wang, Debora Tibbe, Fatemeh Hassani Nia, Antonio Virgilio Failla, Maria Kibæk, Tinett Martesen Overgård, Martin Jakob Larsen, Christina Ringmann Fagerberg, Igor Barsukov, Hans‐Jürgen Kreienkamp

The paper identifies a SHANK3 missense mutation linked to ADHD-like phenotype and shows it disrupts protein interactions critical for neurodevelopmental function.

Scientific Reports · Jan 18, 2022Peer-reviewedNeuroscience