Author

Fatemeh Hassani Nia

University Medical Center Hamburg-EppendorfORCID 0000-0002-5036-0317Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:differential expression· 2 Organism:human· 2 Research Focus:protein structure prediction· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Organism:mouse· 1 Assay:protein structure· 1

Frequent Co-Authors

Hans‐Jürgen KreienkampiD· 3 shared Daniel WoikeiD· 2 shared Debora Tibbe· 2 shared Emily Wang· 2 shared Igor BarsukoviD· 2 shared Alla S. KostyukovaiD· 1 shared Antonio Virgilio FaillaiD· 1 shared Christina Ringmann FagerbergiD· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersNeuroscience

Hans‐Jürgen KreienkampORCID

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintApr 28, 2023

The Shank/ProSAP N-terminal (SPN) domain of Shank3 regulates targeting to postsynaptic sites and postsynaptic signalling.

Daniel Woike, Debora Tibbe, Fatemeh Hassani Nia…·Hans‐Jürgen Kreienkamp

This study investigates how the SPN-Ank domain of Shank3 regulates its localization and function in postsynaptic sites, showing its role in synaptic signaling and implications for autism-related mutations.

1 citation

mouseproteomics

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NeuroscienceScientific ReportsPeer-reviewedJan 18, 2022

Mutations affecting the N-terminal domains of SHANK3 point to different pathomechanisms in neurodevelopmental disorders.

Daniel Woike, Emily Wang, Debora Tibbe…·Hans‐Jürgen Kreienkamp

The paper identifies a SHANK3 missense mutation linked to ADHD-like phenotype and shows it disrupts protein interactions critical for neurodevelopmental function.

15 citations

humanexperimental assay

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NeuroscienceeLifePeer-reviewedMay 4, 2021

Autism associated SHANK3 missense point mutations impact conformational fluctuations and protein turnover at synapses

Michael Bucher, Stephan Niebling, Yuhao Han…·Marina Mikhaylova

This study shows two ASD-associated SHANK3 mutations cause structural changes and altered protein turnover at synapses.

33 citations

humanprotein structure

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