Author

Emily Wang

University of LiverpoolPrimary ORCID profile (0009-0008-9288-6860 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Emily WangiD1 paper · University of Michigan

Biology Disciplines

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Neuroscience· 2 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:differential expression· 2 Organism:human· 2 Research Focus:protein structure prediction· 2 Assay:proteomics· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Daniel WoikeiD· 2 shared Debora Tibbe· 2 shared Fatemeh Hassani NiaiD· 2 shared Hans‐Jürgen KreienkampiD· 2 shared Igor BarsukoviD· 2 shared Antonio Virgilio FaillaiD· 1 shared Arth BankaiD· 1 shared Bailey K. WebsteriD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersNeuroscience

Daniel WoikeORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

RNA-binding proteins activate transcription through defined molecular grammars

Haoran Wang, Lydia Phillips, Dervla Moore-Frederick, Bailey K. Webster, Deyuan Xu, Sid Srivastava, Grant Mowry, Emily Wang, Arth Banka, Changfeng Deng, David M. Chenoweth, Jonathan E. Henninger

The paper identifies RNA-binding proteins (RBPs) that activate transcription through molecular grammars similar to transcription factors, revealing their role in transcriptional regulation and RNA processing.

bioRxiv · May 29, 2026 · PreprintMolecular Biology

The Shank/ProSAP N-terminal (SPN) domain of Shank3 regulates targeting to postsynaptic sites and postsynaptic signalling.

Daniel Woike, Debora Tibbe, Fatemeh Hassani Nia, Victoria Martens, Emily Wang, Igor Barsukov, Hans‐Jürgen Kreienkamp

This study investigates how the SPN-Ank domain of Shank3 regulates its localization and function in postsynaptic sites, showing its role in synaptic signaling and implications for autism-related mutations.

bioRxiv · Apr 28, 2023 · PreprintNeuroscience

Mutations affecting the N-terminal domains of SHANK3 point to different pathomechanisms in neurodevelopmental disorders.

Daniel Woike, Emily Wang, Debora Tibbe, Fatemeh Hassani Nia, Antonio Virgilio Failla, Maria Kibæk, Tinett Martesen Overgård, Martin Jakob Larsen, Christina Ringmann Fagerberg, Igor Barsukov, Hans‐Jürgen Kreienkamp

The paper identifies a SHANK3 missense mutation linked to ADHD-like phenotype and shows it disrupts protein interactions critical for neurodevelopmental function.

Scientific Reports · Jan 18, 2022Peer-reviewedNeuroscience