Author

Dina Alter

University of Virginia

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Organism:Drosophila· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Alissa L. NanaiD· 1 shared Ankit GuptaiD· 1 shared Brandon B. HolmesiD· 1 shared Brandon I. Apresa· 1 shared Bruce L. MilleriD· 1 shared Carlo CondelloiD· 1 shared Indigo V.L. RoseiD· 1 shared Jaeda Coutinho‐BuddiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Alissa L. NanaORCID

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1 paper

Recent·Most cited

β-Amyloid Induces Microglial Expression of GPC4 and APOE Leading to Increased Neuronal Tau Pathology and Toxicity

Brandon B. Holmes, Thaddeus K. Weigel, Jesseca M. Chung, Sarah K. Kaufman, Brandon I. Apresa, James R. Byrnes, Kaan S. Kumru, Jaime Vaquer‐Alicea, Ankit Gupta, Indigo V.L. Rose, Yun Zhang, Alissa L. Nana, Dina Alter, Lea T. Grinberg, Salvatore Spina, , James A. Wells

This study shows that β-amyloid induces microglial GPC4 and APOE, which increases neuronal tau pathology and toxicity.

Molecular Neurodegeneration · Aug 29, 2025Peer-reviewedNeuroscience