Author

E. Balducci

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

A. Bennaceur-Griscelli· 1 shared A. G. Turhan· 1 shared C. Costa· 1 shared C. Desterke· 1 shared E. Macintyre· 1 shared E. Verhoeyen· 1 shared G. Andrieu· 1 shared G. Hypolite· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. Bennaceur-Griscelli

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Modeling Global Genomic Instability in Chronic Myeloid Leukemia (CML) using patient-derived induced pluripotent stem cells (iPSC)

G. Telliam, C. Desterke, J. Imeri, R. Mkacher, N. Oudrhiri, E. Balducci, M. Arnoux, H. Acloque, A. Bennaceur-Griscelli, A. G. Turhan

This study uses patient-derived iPSCs with in vitro mutagenesis to model genomic instability in CML, identifying aberrations linked to blast crisis progression.

Cancers · May 3, 2023Peer-reviewedCancer Biology

ATP citrate lyase is an essential player of the metabolic rewiring induced by PTEN loss during T-ALL development.

G. Andrieu, G. Hypolite, E. Balducci, C. Costa, E. Verhoeyen, I. Nemazanyy, G. Panasyuk, K. Wellen, E. Macintyre, V. Asnafi, M. Tesio

This study identifies ATP citrate lyase (ACLY) as a critical metabolic driver in PTEN-deficient T-ALL, showing its essential role in disease progression and therapeutic targeting potential.

bioRxiv · Mar 27, 2023Cancer Biology