Author

E. Cormet-Boyaka

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1

Frequent Co-Authors

E. Kim· 2 shared P. Boyaka· 2 shared A. Alsudayri· 1 shared A. Amer· 1 shared A. Badr· 1 shared A. Eddy· 1 shared A. Forero· 1 shared A. Kenney· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersImmunology

E. Kim

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Loss of Paneth cells dysregulates gut ILC subsets and enhances weight gain response to high fat diet in a mouse model.

M. R. Joldrichsen, E. Kim, H. E. Steiner, Y. J. Jeong, C. Premanandan, W. Hsueh, O. iouzenkova, E. Cormet-Boyaka, P. Boyaka

The paper shows that loss of Paneth cells in mice alters gut ILC subsets and increases weight gain on a high-fat diet.

bioRxiv · Mar 29, 2024 · PreprintImmunology

MUCOSECRETORY LUNG DISEASE: DIFFERENT ASSEMBLIES OF JAG1 AND JAG2 DETERMINE TRACHEOBRONCHIAL CELL FATE

S. D. Reynolds, C. L. Hill, A. Alsudayri, S. W. Lallier, S. Wijeratne, Z. H. Tan, T. Chiang, E. Cormet-Boyaka

This study shows JAG1 and JAG2 regulate tracheobronchial stem/progenitor cell fate through distinct Notch signaling assemblies, with JAG2 trafficking affecting ligand expression levels.

bioRxiv · Jan 29, 2022 · PreprintCell Biology

Caspase-4/11 exacerbates disease severity in SARS-CoV-2 infection by promoting inflammation and thrombosis

M. Eltobgy, A. Zani, A. Kenney, S. Estfanous, E. Kim, A. Badr, C. Carafice, K. Daily, O. Whitham, M. Pietrzak, A. Webb, J. Kawahara, A. Eddy, P. Denz, M. Lu, , A. Amer

This study shows that caspase-4/11 worsens SARS-CoV-2 infection by increasing inflammation and thrombosis, independent of GSDMD, suggesting it as a potential drug target for severe COVID-19.

bioRxiv · Sep 24, 2021Immunology