Author

E. F. Davis-Marcisak

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1 Approach:deep learning· 1 Approach:experimental assay· 1

Frequent Co-Authors

E. J. Fertig· 2 shared E. M. Jaffee· 2 shared A. A. Fitzgerald· 1 shared A. Gupta· 1 shared B. J. Christmas· 1 shared C. Rafie· 1 shared D. A. Gaykalova· 1 shared D. N. Sidiropoulos· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersImmunology

E. J. Fertig

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2 papers

Recent·Most cited

Phenotypic shifts of tumor associated macrophages and STAT3 mediated suppression of myeloid derived suppressor cells drive sensitization of HER2+ tumor immunity.

D. N. Sidiropoulos, C. Rafie, B. J. Christmas, E. F. Davis-Marcisak, G. Sharma, E. Bigelow, A. Gupta, S. Yegnasubramanian, V. Stearns, R. M. Connolly, D. A. Gaykalova, L. T. Kagohara, E. M. Jaffee, E. J. Fertig, E. T. Roussos-Torres

The paper uses single-cell RNA-sequencing to show entinostat shifts tumor microenvironment cell states, reducing immunosuppression and enhancing immune checkpoint inhibitor responses in HER2+ breast tumors.

bioRxiv · Jan 29, 2021 · PreprintImmunology

Transfer learning between preclinical models and human tumors identifies conserved NK cell activation signature in anti-CTLA-4 responsive tumors

E. F. Davis-Marcisak, A. A. Fitzgerald, M. D. Kessler, L. Danilova, E. M. Jaffee, N. Zaidi, L. M. Weiner, E. J. Fertig

The paper uses transfer learning to identify a conserved NK cell activation signature in anti-CTLA-4 responsive tumors across preclinical models and humans, showing its clinical relevance.

bioRxiv · May 31, 2020 · PreprintCancer Biology