Author

L. M. Weiner

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:deep learning· 1

Frequent Co-Authors

E. J. Fertig· 3 shared A. A. Fitzgerald· 2 shared A. Nasir· 1 shared A. Zuo· 1 shared C. J. Cooper· 1 shared E. F. Davis-Marcisak· 1 shared E. F. Marcisak· 1 shared E. Glasgow· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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3 shared papersCancer Biology

E. J. Fertig

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Fibroblast activation protein regulates natural killer cell migration, extravasation and tumor infiltration

A. A. Fitzgerald, R. E. Maynard, E. F. Marcisak, A. Nasir, E. Glasgow, S. Jablonski, P. Van der Veken, G. Pearson, S. E. Eisman, E. M. Mace, E. J. Fertig, L. M. Weiner

This study shows that fibroblast activation protein (FAP) is expressed by human NK cells and promotes their migration, extravasation, and tumor infiltration, suggesting FAP as a target to enhance NK cell-based therapies.

bioRxiv · Feb 3, 2021 · PreprintImmunology

Immune Selection Pressure Contributes to Pancreatic Cancer Immune Evasion

R. Ajina, A. Zuo, S. Wang, M. Moussa, C. J. Cooper, Y. Shen, Q. R. Johnson, J. M. Parks, J. C. Smith, M. Catalfamo, E. J. Fertig, S. A. Jablonski, L. M. Weiner

This study shows immune selection pressure in pancreatic cancer drives myeloid mimicry and immune evasion, which can be counteracted by JAK/STAT pathway inhibition to enhance immunotherapy.

bioRxiv · Jun 15, 2020 · PreprintCancer Biology

Transfer learning between preclinical models and human tumors identifies conserved NK cell activation signature in anti-CTLA-4 responsive tumors

E. F. Davis-Marcisak, A. A. Fitzgerald, M. D. Kessler, L. Danilova, E. M. Jaffee, N. Zaidi, L. M. Weiner, E. J. Fertig

The paper uses transfer learning to identify a conserved NK cell activation signature in anti-CTLA-4 responsive tumors across preclinical models and humans, showing its clinical relevance.

bioRxiv · May 31, 2020 · PreprintCancer Biology