Author

E. G. Buettmann

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Bioengineering· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:bulk RNA-seq· 1 Research Focus:development & morphogenesis· 1 Research Focus:gene regulatory network inference· 1 Assay:imaging· 1

Frequent Co-Authors

G. A. Hoppock· 2 shared H. J. Donahue· 2 shared M. A. Friedman· 2 shared A. Hensley· 1 shared A. McAlinden· 1 shared A. N. Miller· 1 shared D. A. Sykes· 1 shared D. Skouteris· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersBioengineering

G. A. Hoppock

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Genetic Diversity Modulates The Physical And Transcriptomic Response Of Skeletal Muscle To Simulated Microgravity

Y. Zeineddine, M. A. Friedman, E. G. Buettmann, L. B. Abraham, G. A. Hoppock, H. J. Donahue

This study shows that genetic diversity affects skeletal muscle response to simulated microgravity, with some strains experiencing more muscle loss than others.

bioRxiv · Jun 27, 2023 · PreprintBioengineering

Connexin 43 and Cell Culture Substrate Differentially Regulate OCY454 Osteocytic Differentiation and Signaling to Primary Bone Cells

G. A. Hoppock, E. G. Buettmann, J. A. Denisco, G. M. Goldscheitter, S. N. Condyles, O. J. Juhl, M. A. Friedman, Y. Zhang, H. J. Donahue

This study compares 3D and 2D culture of Cx43-deficient OCY454 osteocytes, finding that 3D cultures enhance differentiation and bone remodeling factor secretion, while Cx43 deficiency increases signaling to promote osteoblast and osteoclast formation.

bioRxiv · Jun 23, 2023Cancer Biology

Ablation of Proliferating Osteoblast Lineage Cells After Fracture Leads to Atrophic Nonunion in a Mouse Model

K. R. Hixon, J. A. McKenzie, D. A. Sykes, S. Yoneda, A. Hensley, E. G. Buettmann, H. Zheng, D. Skouteris, A. McAlinden, A. N. Miller, M. J. Silva

This study shows that ablating proliferating osteoblast lineage cells after fracture in mice leads to atrophic nonunion, providing a new model for studying this condition.

Journal of Bone and Mineral Research · Nov 1, 2021Peer-reviewedBioengineering