Author

A. Hensley

Texas Tech University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Bioengineering· 1 Biophysics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell signaling & trafficking· 2 Research Focus:development & morphogenesis· 1 Organism:Drosophila· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1

Frequent Co-Authors

A. Yildiz· 2 shared A. McAlinden· 1 shared A. N. Miller· 1 shared A. P. Carter· 1 shared A. Vagnoni· 1 shared D. A. Sykes· 1 shared D. Salvador-Garcia· 1 shared D. Skouteris· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersCell Biology

A. Yildiz

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Author profile →

4 papers

Recent·Most cited

Vav2 is a master regulator of repair against bacterial pore-forming toxins.

Gbenga Victor Kayejo, A. Hensley, Tejal Katore, Peter A. Keyel

This study identifies Vav2 as the master regulator of Ca2+-dependent repair against bacterial pore-forming toxins in necrotizing soft tissue infections.

Life Science Alliance · Jul 9, 2026Peer-reviewedCell Biology

A force-sensitive mutation reveals a spindle assembly checkpoint-independent role for dynein in anaphase progression

D. Salvador-Garcia, L. Jin, A. Hensley, M. Golcuk, E. Gallaud, S. Chaaban, F. Port, A. Vagnoni, V. J. Planelles-Herrero, M. A. McClintock, E. Derivery, A. P. Carter, R. Giet, M. Gur, A. Yildiz, S. L. Bullock

This study identifies a mutation in dynein that causes mitotic spindle arrest independent of the spindle assembly checkpoint, revealing a load-dependent role in anaphase progression.

bioRxiv · Aug 3, 2023 · PreprintCell Biology

Mitochondrial TRAK adaptors coordinate dynein and kinesin motility

J. T. Canty, A. Hensley, A. Yildiz

The paper investigates mitochondrial transport in neurons by reconstituting the machinery in vitro, revealing how TRAK adaptors coordinate dynein and kinesin motors and how syntaphilin stalls transport in high Ca2+ regions.

bioRxiv · Jul 30, 2021Biophysics

Ablation of Proliferating Osteoblast Lineage Cells After Fracture Leads to Atrophic Nonunion in a Mouse Model

K. R. Hixon, J. A. McKenzie, D. A. Sykes, S. Yoneda, A. Hensley, E. G. Buettmann, H. Zheng, D. Skouteris, A. McAlinden, A. N. Miller, M. J. Silva

This study shows that ablating proliferating osteoblast lineage cells after fracture in mice leads to atrophic nonunion, providing a new model for studying this condition.

bioRxiv · Oct 6, 2020Bioengineering