Author

E. Goldschmidt

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Organism:human· 2 Assay:scRNA-seq· 2 Approach:statistical model· 2 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Madi· 2 shared A. Barzel· 1 shared A. C. Anderson· 1 shared A. D. Nahmad· 1 shared A. Regev· 1 shared C. Lambden· 1 shared E. Reinstein· 1 shared E. Reuveni· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersImmunology

A. Madi

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Pan-cancer mapping of single T cell profiles reveals a TCF1:CXCR6-CXCL16 regulatory axis essential for effective anti-tumor immunity

L. Jerby, K. Tooley, G. Escobar, G. Dandekar, A. Madi, E. Goldschmidt, C. Lambden, O. Rozenblatt-Rosen, A. C. Anderson, A. Regev

The paper identifies a TCF1:CXCR6-CXCL16 regulatory axis critical for anti-tumor immunity by analyzing single-cell RNA-seq data from multiple cancers.

bioRxiv · Oct 31, 2021 · PreprintImmunology

Frequent Aneuploidy in Primary Human T Cells Following CRISPR-Cas9 cleavage

A. D. Nahmad, E. Reuveni, E. Goldschmidt, T. Tenne, M. Liberman, M. Horovitz-Fried, R. Khosravi, H. Kobo, E. Reinstein, A. Madi, U. Ben-David, A. Barzel

This study finds frequent aneuploidy and chromosomal truncations in CRISPR-Cas9-edited primary human T cells, associated with reduced proliferation and p53 activation.

bioRxiv · Aug 20, 2021Bioengineering