Author

A. C. Anderson

United States Geological Survey

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 5 Biochemistry· 1 Ecology· 1 Zoology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 5 Approach:experimental assay· 5 Organism:mouse· 4 Research Focus:host-pathogen & immune response· 3 Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Research Focus:ecology & biodiversity· 2 Organism:human· 2

Frequent Co-Authors

A. Regev· 3 shared K. Tooley· 3 shared V. K. Kuchroo· 3 shared A. Madi· 2 shared Brock GearyiD· 2 shared D. Mangani· 2 shared Delaina LeBlanc· 2 shared J. Hardin WaddleiD· 2 shared

Activity by Year

2019-2025 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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3 shared papersImmunology

A. Regev

Mutual co-authors in cluster

Author profile →

8 papers

Recent·Most cited

Understanding Abundances and Behaviors of Shorebirds in Coastal Louisiana

Theodore J. Zenzal, A. C. Anderson, Nicholas M. Enwright, Hana R. Thurman, Wyatt C. Cheney, Delaina LeBlanc, Robert C. Dobbs, Brock Geary, J. Hardin Waddle

The paper examines how shorebird abundance and behaviors respond to restoration and habitat factors in coastal Louisiana using remote sensing data and boosted regression tree models.

bioRxiv · Nov 15, 2025 · PreprintEcology

Piping Plover Home Ranges Do Not Appear to be Impacted by Restoration of Barrier Islands and Headlands

Theodore J. Zenzal, A. C. Anderson, Delaina LeBlanc, Robert C. Dobbs, Brock Geary, J. Hardin Waddle

The study examines whether Piping Plover home ranges changed due to barrier island and headland restoration, finding no significant impacts on home range size.

bioRxiv · Mar 3, 2025 · PreprintZoology

Charting the cellular biogeography in colitis reveals fibroblast trajectories and coordinated spatial remodeling

P. Cadinu, K. N. Sivanathan, A. Misra, R. J. Xu, D. Mangani, E. Yang, J. M. Rone, K. Tooley, Y.-C. Kye, L. Bod, L. Geistlinger, T. Lee, N. Ono, G. Wang, L. Sanmarco, , R. Nowarski

The paper uses MERFISH to analyze gene expression in a mouse colitis model, revealing fibroblast trajectories and spatial remodeling during inflammation.

bioRxiv · May 8, 2023Immunology

Pan-cancer mapping of single T cell profiles reveals a TCF1:CXCR6-CXCL16 regulatory axis essential for effective anti-tumor immunity

L. Jerby, K. Tooley, G. Escobar, G. Dandekar, A. Madi, E. Goldschmidt, C. Lambden, O. Rozenblatt-Rosen, A. C. Anderson, A. Regev

The paper identifies a TCF1:CXCR6-CXCL16 regulatory axis critical for anti-tumor immunity by analyzing single-cell RNA-seq data from multiple cancers.

bioRxiv · Oct 31, 2021 · PreprintImmunology

Induction of a colitogenic phenotype in Th1 cells depends on IL-23R signaling

M. Pawlak, D. DeTomaso, G. Meyer zu Horste, Y. Lee, J. Nyman, D. Dionne, C. Wang, A. Wallrapp, P. R. Burkett, S. J. Riesenfeld, A. C. Anderson, A. Regev, R. J. Xavier, N. Yosef, V. K. Kuchroo

This study shows IL-23R signaling enables Th1 cells to acquire a colitogenic phenotype, distinct from Th17 cells, and identifies CD160 as a key regulator.

bioRxiv · Jan 24, 2021 · PreprintImmunology

Identification of the Clostridial cellulose synthase and characterization of the cognate glycosyl hydrolase, CcsZ

W. Scott, B. Lowrance, A. C. Anderson, J. Weadge

This paper identifies a gene cluster encoding a cellulose synthase in Clostridia and characterizes the glycosyl hydrolase CcsZ as an endo-acting cellulase.

bioRxiv · Nov 10, 2019Biochemistry

An endogenous glucocorticoid-cytokine signaling circuit promotes CD8+ T cell dysfunction in the tumor microenvironment

N. Acharya, A. Madi, H. Zhang, M. Klapholz, E. Christian, K. O. Dixon, G. Fell, K. Tooley, D. Mangani, J. Xia, M. Singer, D. Neuberg, O. Rozenblatt-Rosen, A. Regev, V. K. Kuchroo, A. C. Anderson

This study identifies a glucocorticoid-cytokine signaling circuit in the tumor microenvironment that promotes CD8+ T cell dysfunction and correlates with poor response to checkpoint blockade in melanoma.

bioRxiv · Oct 10, 2019 · PreprintImmunology

Male sex chromosomal complement exacerbates the pathogenicity of Th17 cells in a chronic model of CNS autoimmunity.

P. M. I. A. Doss, J. Baillargeon, A. P. Yeola, J. B. Williams, M. Leclercq, C. Joly-Beauparlant, M. Alpaugh, A. C. Anderson, A. Droit, H. Lassmann, C. S. Moore, M. Rangachari

This study shows male sex chromosomal complement increases Th17 cell pathogenicity in CNS autoimmunity, independent of hormones, through downregulation of Jarid1c.

bioRxiv · Jun 13, 2019 · PreprintImmunology