Author

E. M. Saraiva

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:flow cytometry· 1

Frequent Co-Authors

A. A. Dias· 1 shared A. A. S. Pinheiro· 1 shared A. Dicko· 1 shared A. Machado· 1 shared C. Canetti· 1 shared C. F. Benjamim· 1 shared C. M. Takiya· 1 shared C. Mermelstein· 1 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperImmunology

A. A. Dias

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Author profile →

3 papers

Recent·Most cited

Adenosine diphosphate contributes to wound healing in diabetic mice through P2Y1 and P2Y12 receptors activation

P. A. Borges, I. Waclawiak, J. L. Georgii, J. F. Barros, V. S. Fraga-Junior, F. S. Lemos, T. Russo-Abrahao, E. M. Saraiva, C. M. Takiya, R. Coutinho-Silva, C. Penido, C. Mermelstein, J. R. Meyer-Fernandes, F. Canto, J. S. Neves, , C. F. Benjamim

This study shows adenosine diphosphate accelerates wound healing in diabetic mice by activating P2Y1 and P2Y12 receptors, enhancing tissue repair and reducing inflammation.

bioRxiv · Oct 22, 2020Immunology

CXCR4 and MIF are required for neutrophil extracellular trap release triggered by Plasmodium-infected erythrocytes

D. S. A. Rodrigues, E. B. Prestes, L. d. S. Silva, A. A. S. Pinheiro, J. M. C. Ribeiro, A. Dicko, P. E. Duffy, M. Fried, I. M. B. Francischetti, E. M. Saraiva, H. A. Paula-Neto, M. T. Bozza

The paper shows that Plasmodium-infected erythrocytes release MIF, which triggers CXCR4-dependent NET formation by neutrophils, contributing to parasite control in malaria.

bioRxiv · Nov 22, 2019Immunology

Neutrophil Extracellular Traps contribute to the pathogenesis of leprosy type 2 reactions

C. O. da Silva, A. A. Dias, J. A. Nery, A. Machado, H. Ferreira, J. P. Santos, N. Nadaes, E. N. Sarno, E. M. Saraiva, V. Schmitz, M. C. V. Pessolani

This study investigates the role of neutrophil extracellular traps (NETs) in the pathogenesis of leprosy type 2 reactions and shows that thalidomide treatment reduces NET formation.

bioRxiv · Apr 8, 2019Immunology