Author

J. M. C. Ribeiro

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Assay:flow cytometry· 1 Organism:human· 1

Frequent Co-Authors

A. A. S. Pinheiro· 1 shared A. Adegoke· 1 shared A. B. F. Barletta· 1 shared A. Dicko· 1 shared C. Barillas-Mury· 1 shared C. Meneses· 1 shared D. S. A. Rodrigues· 1 shared E. B. Prestes· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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1 shared paperImmunology

A. A. S. Pinheiro

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Rickettsia parkeri Hijacks Tick Hemocytes to Manipulate Cellular and Humoral Transcriptional Responses

A. Adegoke, J. M. C. Ribeiro, S. Brown, R. C. Smith, S. Karim

This study identifies five hemocyte populations in ticks and shows Rickettsia parkeri infects phagocytic hemocytes, altering immune gene expression and phagocytosis.

bioRxiv · Nov 9, 2022 · PreprintImmunology

Genetic exchange in Leishmania is facilitated by IgM natural antibodies

T. D. Serafim, E. Iniguez, A. B. F. Barletta, J. S. P. Doehl, M. Short, J. Lack, P. Cecilio, V. Nair, M. Disotuar, T. Wilson, I. V. Coutinho-Abreu, F. Oliveira, C. Meneses, C. Barillas-Mury, J. Andersen, , J. G. Valenzuela

This study shows IgM antibodies facilitate Leishmania genetic hybridization in vitro and in vivo, leading to full genome hybrids and increased genetic exchange.

bioRxiv · Jun 9, 2022 · PreprintMicrobiology

CXCR4 and MIF are required for neutrophil extracellular trap release triggered by Plasmodium-infected erythrocytes

D. S. A. Rodrigues, E. B. Prestes, L. d. S. Silva, A. A. S. Pinheiro, J. M. C. Ribeiro, A. Dicko, P. E. Duffy, M. Fried, I. M. B. Francischetti, E. M. Saraiva, H. A. Paula-Neto, M. T. Bozza

The paper shows that Plasmodium-infected erythrocytes release MIF, which triggers CXCR4-dependent NET formation by neutrophils, contributing to parasite control in malaria.

bioRxiv · Nov 22, 2019Immunology