Author

E. Yamasaki

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:development & morphogenesis· 1 Assay:electrophysiology· 1 Approach:genetic perturbation / CRISPR· 1 Assay:imaging· 1 Approach:microscopy & imaging analysis· 1

Frequent Co-Authors

P. Thakore· 2 shared S. Ali· 2 shared S. Earley· 2 shared A. L. Gonzales· 1 shared A. S. Solano· 1 shared C. Labelle-Dumais· 1 shared C. S. Griffin· 1 shared D. B. Gould· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersPathology

P. Thakore

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Author profile →

2 papers

Recent·Most cited

Impaired intracellular Ca2+ signaling contributes to age-related cerebral small vessel disease in Col4a1 mutant mice

E. Yamasaki, P. Thakore, S. Ali, A. S. Solano, X. Wang, X. Gao, C. Labelle-Dumais, M. M. Chaumeil, D. B. Gould, S. Earley

This study identifies impaired SR Ca2+ signaling and disrupted ion channel activity in Col4a1 mutant mice as contributors to age-related cerebral small vessel disease.

bioRxiv · Aug 24, 2022 · PreprintPathology

Peripheral Coupling Sites Formed by STIM1 Govern the Contractility of Vascular Smooth Muscle Cells

V. Krishnan, S. Ali, A. L. Gonzales, P. Thakore, C. S. Griffin, E. Yamasaki, M. G. Alvarado, M. T. Johnson, M. Trebak, S. Earley

This study shows that STIM1 maintains peripheral coupling in vascular smooth muscle cells, essential for contractility, independent of ER Ca2+ store depletion.

bioRxiv · May 25, 2021Physiology