Author

S. Earley

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 1 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:development & morphogenesis· 1 Assay:electrophysiology· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1

Frequent Co-Authors

E. Yamasaki· 2 shared P. Thakore· 2 shared S. Ali· 2 shared A. Ahkter· 1 shared A. Allue Guardia· 1 shared A. Garcia Vilanova· 1 shared A. L. Gonzales· 1 shared A. Olmo-Fontanez· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersPathology

E. Yamasaki

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Impaired intracellular Ca2+ signaling contributes to age-related cerebral small vessel disease in Col4a1 mutant mice

E. Yamasaki, P. Thakore, S. Ali, A. S. Solano, X. Wang, X. Gao, C. Labelle-Dumais, M. M. Chaumeil, D. B. Gould, S. Earley

This study identifies impaired SR Ca2+ signaling and disrupted ion channel activity in Col4a1 mutant mice as contributors to age-related cerebral small vessel disease.

bioRxiv · Aug 24, 2022 · PreprintPathology

Peripheral Coupling Sites Formed by STIM1 Govern the Contractility of Vascular Smooth Muscle Cells

V. Krishnan, S. Ali, A. L. Gonzales, P. Thakore, C. S. Griffin, E. Yamasaki, M. G. Alvarado, M. T. Johnson, M. Trebak, S. Earley

This study shows that STIM1 maintains peripheral coupling in vascular smooth muscle cells, essential for contractility, independent of ER Ca2+ store depletion.

bioRxiv · May 25, 2021Physiology

Lethality of SARS-CoV-2 infection in K18 human angiotensin converting enzyme 2 transgenic mice

F. Oladunni, J.-G. Park, P. Pino Tamayo, O. Gonzalez, A. Ahkter, A. Allue Guardia, A. Olmo-Fontanez, S. Gautam, A. Garcia Vilanova, C. Ye, K. Chiem, C. Headley, V. Dwivedi, L. Parodi, K. Alfson, , J. B. Torrelles

This study demonstrates that K18 hACE2 transgenic mice are susceptible to SARS-CoV-2 infection, exhibit cytokine and chemokine storms, and serve as a model for studying viral pathogenesis and therapeutic development.

bioRxiv · Jul 18, 2020Microbiology