Author

Elda M. Munivez

Baylor College of Medicine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Genetics· 2 Systems Biology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:differential expression· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:proteomics· 1 Organism:zebrafish· 1

Frequent Co-Authors

Brendan LeeiD· 2 shared Catherine Glauber AmbroseiD· 2 shared Cole D KuzawaiD· 2 shared Abbey A. ScottiD· 1 shared Alistair Duncan CalderiD· 1 shared Alyssa A. Tran· 1 shared Anna MalovannayaiD· 1 shared Antrix JainiD· 1 shared

Activity by Year

2020-2024 · peak 1/yr

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10 top collaborators · 29 mutual co-author links

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2 shared papersGenetics

Brendan LeeORCID

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3 papers

Recent·Most cited

Atrial Proteomic Profiling Reveals a Switch Towards Profibrotic Gene Expression Program in CREM-IbΔC-X Mice with Persistent Atrial Fibrillation

Shuai Zhao, Mohit M. Hulsurkar, Satadru K. Lahiri, Yuriana Aguilar-Sánchez, Elda M. Munivez, Frank U. Müller, Antrix Jain, Anna Malovannaya, Chi Him Kendrick Yiu, Svetlana N. Reilly, Xander H.T. Wehrens

This study identifies proteomic changes in CREM-IbΔC-X transgenic mice with persistent atrial fibrillation, showing a shift from metabolic to profibrotic gene expression programs with age.

Journal of Molecular and Cellular Cardiology · May 1, 2024Peer-reviewedSystems Biology

Tendon and Motor Phenotypes in the Crtap-/- Mouse Model of Recessive Osteogenesis Imperfecta

Matthew William Grol, Nele A.T Haelterman, Joohyun Lim, Elda M. Munivez, Marilyn Archer, David M. Hudson, Sara F. Tufa, Douglas R. Keene, Kevin Lei, Dongsu Park, Cole D Kuzawa, Catherine Glauber Ambrose, David R. Eyre, Brendan Lee

This study examines tendon and motor phenotypes in Crtap-/- mice, finding thinner tendons, altered collagen structure, and motor impairments linked to tendon/ligament dysfunction in OI.

eLife · May 26, 2021Peer-reviewedGenetics

COPB2 haploinsufficiency causes a coatopathy with osteoporosis and developmental delay

Ronit Marom, Lindsay Catherine Burrage, Aurélie Clément, Bernardo Blanco‐Sánchez, Rossella Venditti, Mahim Jain, Ingo Grafe, Daryl A. Scott, Jill Anne Rosenfeld, Vernon Reid Sutton, Marwan S. Shinawi, Ghayda Mirzaa, Catherine J DeVile, Rowenna Roberts, Alistair Duncan Calder, , Brendan Lee

This study identifies COPB2 haploinsufficiency as a cause of a coatopathy with osteoporosis and developmental delay, showing disrupted collagen trafficking and potential treatment with ascorbic acid.

bioRxiv · Sep 14, 2020 · PreprintGenetics