Author

F. Aulicino

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:fluorescence / live-cell microscopy· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

E. Pedone· 1 shared F. Cottaz· 1 shared F. Lluis· 1 shared F. Sottile· 1 shared I. Berger· 1 shared L. Marucci· 1 shared M. P. Cosma· 1 shared M. Pelosse· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCell Biology

E. Pedone

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2 papers

Recent·Most cited

FRET-based sensor for measuring adenine nucleotide binding to AMPK

R. Abi Nahed, M. Pelosse, F. Aulicino, F. Cottaz, I. Berger, U. Schlattner

The paper describes an AMPK-based FRET sensor, AMPfret, that detects conformational changes in AMPK upon nucleotide binding, enabling analysis of AMPK activation in vitro and in cells.

bioRxiv · Sep 5, 2023 · PreprintBiochemistry

Canonical Wnt pathway controls mESCs self-renewal through inhibition of spontaneous differentiation via β-catenin/TCF/LEF functions

F. Aulicino, F. Sottile, E. Pedone, F. Lluis, L. Marucci, M. P. Cosma

This study shows that canonical Wnt signaling inhibits mESC differentiation by β-catenin/TCF/LEF functions, and that non-functional β-catenin isoforms can rescue self-renewal defects.

bioRxiv · Jun 16, 2019Cell Biology