Author

Felix Hagelskamp

Ludwig-Maximilians-Universität München

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:protein structure prediction· 1 Approach:statistical model· 1

Frequent Co-Authors

Bennett HenzeleriD· 1 shared Dragony FuiD· 1 shared Eric M. PhizickyiD· 1 shared Felix XuiD· 1 shared Florian SchelteriD· 1 shared Fowzan Sami AlkurayaiD· 1 shared Jillian RamosiD· 1 shared Ken Kögel· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperBiochemistry

Bennett HenzelerORCID

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2 papers

Recent·Most cited

ALKBH1 activity in vitro and human cell lines by isotope dilution mass spectrometry

Bennett Henzeler, Olga Hofmeister, Ken Kögel, Ying Qi, Felix Hagelskamp, Matthias Heiß, Florian Schelter, Felix Xu, Lena Josefine Daumann, Thomas Carell, Stefanie Kaiser, Sabine Schneider

This study assesses ALKBH1 activity in vitro and in human cell lines using mass spectrometry, finding no significant impact on tRNA modifications under optimal conditions.

PLoS ONE · Apr 6, 2026Peer-reviewedBiochemistry

Formation of tRNA wobble inosine in humans is perturbed by a millennia-old mutation linked to intellectual disability

Jillian Ramos, Lu Han, Yan Li, Felix Hagelskamp, Stefanie M. Kellner, Fowzan Sami Alkuraya, Eric M. Phizicky, Dragony Fu

This study shows that a mutation in ADAT3, linked to intellectual disability, disrupts tRNA wobble inosine formation and ADAT2/ADAT3 complex function.

Molecular and Cellular Biology · Jul 1, 2019Peer-reviewedMolecular Biology