Author

G. Agnetti

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:proteomics· 3 Research Focus:cell type annotation· 2 Research Focus:protein folding & dynamics· 2 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

H. Kadioglu· 2 shared A. Lyashkov· 1 shared A. Parveen· 1 shared C. G. Glabe· 1 shared C. Morrell· 1 shared D. Riordon· 1 shared E. Lakatta· 1 shared G. Keceli· 1 shared

Activity by Year

2023-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersCell Biology

H. Kadioglu

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3 papers

Recent·Most cited

Age-associated failure of efficient cardiac proteostatic adaptations to chronic cAMP-stress is associated with accelerated heart aging

M. G. Perino, M. Calvo-Rubio Barrera, D. Riordon, G. Agnetti, K. Tarasov, A. Parveen, K. Chakir, C. Morrell, I. Ahmet, A. Lyashkov, Y. Lukyanenko, Y. Tarasova, J. Qu, H. Kadioglu, M. Ranek, , E. Lakatta

This study shows that aging impairs proteostatic mechanisms in mice with cardiac-specific over-expression of adenylyl cyclase VIII, leading to accelerated heart aging and cardiomyopathy.

bioRxiv · Aug 13, 2023 · PreprintCell Biology

Thioflavin T In-gel Stain to Study Protein Misfolding in Frozen Tissue Specimens

J. Oldam, I. Tchernyshyov, J. Van Eyk, J. Troncoso, C. G. Glabe, G. Agnetti

The paper introduces a Thioflavin T in-gel staining method for quantifying and characterizing amyloid species in frozen tissue samples, highlighting its simplicity, cost-effectiveness, and compatibility with downstream analyses.

bioRxiv · May 12, 2023 · PreprintBiochemistry

Ischemia/Reperfusion Injury and Oxidative Stress Impair Cardiac Desmin Proteostasis

Z. Li, S. Jun, K. K. Singh, P. J. Calhoun, G. Keceli, K. Patel, H. Kadioglu, N. Paolocci, G. Agnetti

This study investigates how oxidative stress and reperfusion injury affect desmin proteostasis, identifying mechanisms to reduce toxic desmin aggregates and improve cardiac function.

bioRxiv · May 9, 2023 · PreprintBiochemistry