Author

G. de Carcer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

A. Alonso de la Vega· 1 shared A. de Martino· 1 shared A. El Bakkali· 1 shared A. Monfort-Vengut· 1 shared A. Teijo· 1 shared B. Escobar· 1 shared B. Ortigosa· 1 shared C. Bertinetti· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

A. Alonso de la Vega

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2 papers

Recent·Most cited

TPX2 expression promotes sensitivity to dasatinib in breast cancer by activating the YAP transcriptional signaling.

C. Marugan, B. Ortigosa, N. Sanz-Gomez, A. Monfort-Vengut, C. Bertinetti, A. Teijo, M. Gonzalez, A. Alonso de la Vega, M. J. Lallena, G. Moreno-Bueno, G. de Carcer

The paper identifies TPX2 as a gene that increases breast cancer sensitivity to dasatinib by activating the YAP pathway, and shows TPX2 and YAP are biomarkers for poor prognosis and potential dasatinib therapy.

bioRxiv · Sep 4, 2023Cancer Biology

Plk1 overexpression suppresses tumor development by inducing chromosomal instability

G. de Carcer, S. V. Venkateswaran, L. Salgueiro, A. El Bakkali, K. Somogyi, K. Rowald, P. Montanes, M. Sanclemente, B. Escobar, A. de Martino, N. McGranahan, M. Malumbres, R. Sotillo

This study shows that Plk1 overexpression suppresses tumor development by causing chromosomal instability and cytokinesis defects, despite its role as an oncogene.

bioRxiv · Mar 20, 2018Cancer Biology