Author

B. Escobar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1

Frequent Co-Authors

A. de Martino· 1 shared A. El Bakkali· 1 shared B. Palacios· 1 shared C. Diaz-Diaz· 1 shared G. de Carcer· 1 shared I. Menendez-Montes· 1 shared J. Ruiz-Cabello· 1 shared K. Rowald· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

A. de Martino

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2 papers

Recent·Most cited

Activation of HIF2 leads to vascular remodeling and inflammation, coronary thrombosis and arterial dilation, recapitulating cardiac involvement of Kawasaki disease.

B. Escobar, I. Menendez-Montes, T. Albendea-Gomez, S. Mendoza-Tamajon, R. Castro-Mecinas, C. Diaz-Diaz, B. Palacios, J. Ruiz-Cabello, L. J. Jimenez-Borreguero, M. C. Cid, K. Takahashi, S. Martin-Puig

This study shows that activating HIF2 causes vascular remodeling, inflammation, and coronary issues similar to Kawasaki disease, using a new mouse model and human tissue analysis.

bioRxiv · Jan 22, 2024 · PreprintPathology

Plk1 overexpression suppresses tumor development by inducing chromosomal instability

G. de Carcer, S. V. Venkateswaran, L. Salgueiro, A. El Bakkali, K. Somogyi, K. Rowald, P. Montanes, M. Sanclemente, B. Escobar, A. de Martino, N. McGranahan, M. Malumbres, R. Sotillo

This study shows that Plk1 overexpression suppresses tumor development by causing chromosomal instability and cytokinesis defects, despite its role as an oncogene.

bioRxiv · Mar 20, 2018Cancer Biology