Author

G. Dubyak

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Approach:microscopy & imaging analysis· 1 Organism:mouse· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

A. Rietsch· 1 shared B. A. Miller· 1 shared B. Sahoo· 1 shared E. Pearlman· 1 shared J. Tran-Chau· 1 shared K. Liboro· 1 shared M. Marshall· 1 shared M. Minns· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperMicrobiology

A. Rietsch

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

How NINJ1 mediates plasma membrane rupture and why NINJ2 cannot

B. Sahoo, Z. Mou, W. Liu, G. Dubyak, X. Dai

The paper identifies NINJ1 as an active mediator of plasma membrane rupture and explains why NINJ2 cannot, through structural and functional analysis of their filaments.

bioRxiv · May 31, 2023Immunology

The NLRP3 inflammasome selectively drives IL-1β secretion by Pseudomonas aeruginosa infected neutrophils and regulates bacterial killing in vivo

M. Minns, T. Lima, K. Liboro, S. Abbondante, B. A. Miller, M. Marshall, J. Tran-Chau, A. Rietsch, G. Dubyak, E. Pearlman

This study shows that Pseudomonas aeruginosa uses ExoS to redirect neutrophil inflammasome signaling from NLRC4 to NLRP3, enhancing bacterial killing and reducing disease severity in mice.

bioRxiv · Apr 7, 2022 · PreprintMicrobiology